THE Trauma of voices. Body Mentalists.

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Neurons on Ketones.
Excitory neurons transmit signals and process information, but if excitory neurons become over-stimulated they tend to die. As the same can be said for Traumatic Pathogens of the trauma of mental illness.

The brain excitation and inhibition initializes through two main neurotransmitters, the excitatory Glutamate and the inhibitory GABA. Glutamate in larger quantities causes neurodegeneration, as well as excitotoxicity. Put another way hyper-activity is toxic. The acidity of ketones may decrease the pH of certain brain microdomains, which might be the mechanism of keto’s positive effect on Type II Bipolar disorder. Put another way the Traumatic Psychosocial Pathogens have been dissolved.

http://www.tandfonline.com/doi/full/10.1080/13554794.2012.690421#abstract
Abstract from the above website.

Neurocase: The Neural Basis of Cognition

Volume 19, Issue 5, 2013

Successful mood stabilizing treatments reduce intracellular sodium in an activity-dependent manner. This can also be achieved with acidification of the blood, as is the case with the ketogenic diet. Two women with type II bipolar disorder were able to maintain ketosis for prolonged periods of time (2 and 3 years, respectively). Both experienced mood stabilization that exceeded that achieved with medication; experienced a significant subjective improvement that was distinctly related to ketosis; and tolerated the diet well. There were no significant adverse effects in either case. These cases demonstrate that the ketogenic diet is a potentially sustainable option for mood stabilization in type II bipolar illness. They also support the hypothesis that acidic plasma may stabilize mood, perhaps by reducing intracellular sodium and calcium.

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